Hormones {adrenal corticoid hormone}| can regulate kidney Na+ and K+ reabsorption.
Kidney hormones {aldosterone}| can control blood pressure.
Hormones {antidiuretic hormone, endocrine}| (ADH) can increase kidney water reabsorption and so block water loss.
Mammal hypothalamus supraoptic and paraventricular nuclei synthesize octapeptides or nonapeptides {arginine vasopressin}| (AVP).
functions
AVP regulates water balance. Decreased blood volume or increased plasma osmotic pressure causes AVP secretion. AVP causes blood-vessel constriction, maintaining blood pressure in cases of decreased blood volume. AVP stimulates intestinal motility, lowering fluid loss. AVP increases cell permeability to water in kidney collecting tubules. AVP enhances sodium-chloride active transport in renal medullary tubules.
AVP secretes in pain and stress. AVP stimulates adrenocorticotropic hormone release, triggering adrenal steroid secretions and stress responses.
AVP can affect mammal pair bonding and infant care.
receptors
AVP binds to kidney-tubule, vascular smooth-muscle, pituitary, and intestinal cell receptors.
receptors: baroreceptor
Reduced blood volume decreases blood pressure and stimulates low-pressure stretch baroreceptors in left atrium, aorta, and carotid. Baroreceptors stimulate glossopharyngeal and vagus nerves to hypothalamus, which liberates AVP from pituitary nerve terminals.
receptors: osmoreceptor
Increased plasma concentration and higher osmolality stimulate osmoreceptors in hypothalamus, resulting in AVP secretion.
Hormones {mineralcorticoid} can regulate salts.
Hormones {posterior pituitary neurohormone} can act directly on kidneys, to decrease urine formation and water loss.
Hormones {prodynorphin} can act on posterior pituitary hormones, to control blood volume and regulate blood pressure.
4-Zoology-Organ-Endocrine Gland-Hormone
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Date Modified: 2022.0225